glutamate

Converse of object

• call: It acts by blocking a messenger chemical called glutamate which is produced by the brain cells damaged by the disease.
• use: Comment The evidence for using glutamate is not convincing.
• release: Glioma tumor cells release the neurotransmitter glutamate, which carves a path of destruction through brain tissue allowing the tumor cells to advance.
• activate: The role of glutamate activated signaling pathways in cortical Peter Kind graduated from Dalhousie University with a B.S. in Neurosciences in 1988.
• remove: The drugs increase the number of transporters, chemicals that remove glutamate from nerves.

Preposition: in

• brain: The enzyme is important because it helps remove glutamate in the brain.

Adjective modifier

• e621: The four additives were: E133 brilliant blue; E104 quinoline yellow; E621 monosodium glutamate ( MSG ) and E951 aspartame.
• much: In MS there is evidence that people may be producing too much glutamate.
• excitatory: The role of the excitatory neurotransmitter glutamate is being investigated.

Modifies a noun

• eaat2: Polymorphisms in the glutamate transporter gene EAAT2 in European ALS patients.
• transporter: Polymorphisms in the glutamate transporter gene EAAT2 in European ALS patients.
• dehydrogenase: Glutamate Dehydrogenase catalyzes a major reaction that effects net removal of N from the amino acid pool.
• receptor: All of these mice have a deletion of a particular gene involved in glutamate receptor signaling.
• antagonist: They also used quantal analysis to show that certain glutamate antagonists act solely post-synaptically.
• residue: For example, a glutamate residue may be stabilizing a positively charged intermediate.

Noun used with modifier

• monosodium: This makes for a difficult choice between monosodium glutamate overload or the monthly credit card bill nightmare.
• neurotransmitter: The main culprit has been thought to be the neurotransmitter glutamate acting through AMPA receptors.
• ionotropic: We are currently focussed on two distinct families of these receptors: 1. The ionotropic glutamate receptors and 2. The nicotinic acetylcholine receptor.
• glial: Molecular genetic studies in motor neuron disease: search for interactors with the glial glutamate transporter, EAAT2.
• transmitter: Connections shown in red have an excitatory effect on target structures ( primarily through the transmitter glutamate ).